Home

Asymmetric septal hypertrophy newborn

Riesenauswahl an Markenqualität. Folge Deiner Leidenschaft bei eBay! Über 80% neue Produkte zum Festpreis; Das ist das neue eBay. Finde ‪New Born‬! Abstract The objective of this paper is to determine the frequency and outcome of asymmetrical septal hypertrophy (ASH) in large-for-gestational-age infants (LGA) born to diabetic (DM) and nondiabetic mothers (NDM), and to establish the relationship between ASH and maternal diabetes control Abstract Severe hypertrophy of the interventricular septum was found by echocardiography in a 15 day old infant without symptoms whose mother was not diabetic. The electrocardiogram showed pronounced intraventricular conduction disturbances Asymmetric septal hypertrophy is a common finding in infants born to diabetic mothers, even if they are appropriate for gestational age. Asymmetric septal hypertrophy is a common finding in infants born to diabetic mothers, even if they are appropriate for gestational age 1. Int J Cardiol. 2000 Sep 15;75(2-3):297-8. Asymmetric septal hypertrophy in an infant exposed to ritodrine in utero. Ocal B, Aydin OF, Oguz D, Karademir S

Große Auswahl an ‪New Born - New born

Asymmetrical septal hypertrophy is a common finding in infants of diabetic mothers, with an echocardiographic incidence of 38.8% in this study. The condition occurs independently of maternal. Septal hypertrophy was defined as a septal thickness of 6 mm or greater; a normal newborn infant has a septal thickness of 3 to 4 mm.'2' RESULTS Disproportionate interventricular septal hypertrophy with respect to the left ventricular wall was demonstrated by echocardiography in five of the 18 IDM and in the infant with nesidioblastosis (Fig. 1) Asymmetric hypertrophy of the ventricular septum relative to the left ventricular free wall was present in the 16 patients who had echocardiographic or necropsy examination. Ventricular septal thickening was substantial in patients studied both before and after 6 months of age (mean 16 mm), indicating that in patients with hypertrophic. Asymmetric septal hypertrophy is the most common type of hypertrophic cardiomyopathy in which the abnormal ventricular muscle thickening is confined to the interventricular septum, causing the walls of the lower heart chambers (typically the left ventricle) to become thick and stiff 1)

Cardiomegaly is common (30%), and heart failure occurs in 5-10% of infants of diabetic mothers. Asymmetric septal hypertrophy may occur and become manifested similar to idiopathic hypertrophic subaortic stenosis. d) Hematologic system. Polycythemi Hypertrophic cardiomyopathy often goes undiagnosed because many people with the disease have few, if any, symptoms and can lead normal lives with no significant problems. However, in a small number of people with HCM , the thickened heart muscle can cause shortness of breath, chest pain or problems in the heart's electrical system, resulting in. We report a neonate that was diagnosed as case of asymmetrical septal hypertrophy and hypertrophic cardiomyopathy and was born to mother who had poor glycemic control and was treated successfully with propranolol and showed resolution of hypertrophy in follow-up echocardiography

Asymmetrical septal hypertrophy in newborn infants of diabetic mothers. American journal of perinatology, 17(02), 089-094. Marian, A. J. (2010). Hypertrophic cardiomyopathy: from genetics to treatment in the first 24 hours after birth. Results: Asymmetric septal hypertrophy was only present in infants born to diabetic mothers (50% vs. 0%; P<0.001). Intraventricular septum thickness and intraventricular septum/posterior wall of the left ventricle ratio was also significantly higher in the first group (P<0.001). We foun S. Cardiac septal hypertrophy in hyperinsulinemic infant. J Pediatr. 1980;96:535-9. 3. Cooper MJ, Enderlein MA, Tarnoff H, Rogé CL. Asymmetric septal hypertrophy in infants of diabetic mothers. Am J Dis Child. 1992;146:226-9. 4. Vela-Huerta MM, Vargas-Origel A, Olvera-López A. Asymmetrical septal hypertrophy in newborn infants of diabetic. Asymmetric septal hypertrophy was documented in one first degree relative of each infant. It is concluded that asymmetric septal hypertrophy is a genetically transmitted disease that may present clinically in infancy and lead to infant death. Furthermore, the characteristic pathologic feature of asymmetric septal hypertrophy in adults, a.

To compare the frequency of asymmetric septal hypertrophy in appropriate for gestational age infants born to diabetic mothers with those born to non-diabetic mothers. We compared 38 full term infants born to diabetic mothers with 85 full term infants of non-diabetic mothers. 2-D echocardiography was obtained in the first 24 hours after birth Maternal glycosylated hemoglobin levels were higher during the third trimester in mothers of affected infants. Our data support a possible relationship between third-trimester maternal hyperglycemia and neonatal asymmetric septal hypertrophy, macrosomia, and hypoglycemia. (AJDC. 1992;146:226-229 Familial hypertrophic cardiomyopathy (HCM) is caused by mutations in any of several known genes, and possibly other genes that have not yet been identified. Genetic testing for HCM is most informative as a family test rather than a test of one person. Results are most accurately interpreted after merging both genetic and medical test results from multiple family members The incidence of symptomatic hypertrophic cardiomyopathy is reported to be as high as 12.1% of infants with diabetic mothers 2. Other associated findings include left ventricular outflow tract obstruction and reduced stroke volume 4. Septal enlargement and hypertrophy are secondary to fetal hyperinsulinemia

Pathological left ventricular hypertrophy, asymmetrical septal hypertrophy and outflow tract obstruction is a rare but known cardiac comorbidity in infants of diabetic mothers Similarly the fetal heart, rich in insulin receptors, may undergo increased ventricular septal thickening in the presence of high insulin levels, culminating in asymmetric septal hypertrophy , , . Despite tight control of maternal blood glucose levels in gestational diabetics, asymmetric septal hypertrophic cardiomyopathy (HCM) is still. 22 Vela-Huerta MM, Vargas-Origel A, Olvera-López A. Asymmetrical septal hypertrophy in newborn infants of diabetic mothers. Am J Perinatol 2000;17(02):89-94 [ Links ] 23 Behle I, Zielinsky P, Zimmer LP, Pontremoli M, Risch JN. [Glycosylated hemoglobin levels and cardiac abnormalities in fetuses of diabetic mothers] The obstructive variant of HCM is hypertrophic obstructive cardiomyopathy (HOCM), also historically known as idiopathic hypertrophic subaortic stenosis (IHSS) or asymmetric septal hypertrophy (ASH). Another, non-obstructive variant of HCM is apical hypertrophic cardiomyopathy (AHC), also called Yamaguchi syndrome In patients with asymmetric septal hypertrophy there may be coexisting dynamic left ventricular outflow obstruction and/or systolic anterior motion of mitral valve resulting in mitral regurgitation. Echocardiographic findings of neonatal cardiomyopathies are listed in Table 42.3 and some examples are shown in Figure 42.3

Asymmetrical septal hypertrophy in newborn infants of

Sir.—Cooper et al 1 presented the results of their excellent study correlating echocardiographic findings of fetuses and infants of diabetic mothers with indexes of maternal diabetic control. Unfortunately, they failed to provide enough information on the echocardiographic data to allow assessment of the accuracy of their calculation of 31% prevalence of asymmetric septal hypertrophy (ASH) Transient asymmetric ventricular septal hypertrophy in the newborn unassociated with maternal diabetes. (PMID:6540114 PMCID:PMC481614) PMID:6540114 PMCID:PMC48161

The degree and distribution of LVH is variable: mild hypertrophy (13-15 mm) or extreme myocardial thickening (30-60 mm) may be seen. The most commonly observed pattern is asymmetrical thickening of the anterior interventricular septum (= asymmetrical septal hypertrophy). This pattern has been classically associated with systolic anterior motion. An atrial septal defect will cause a shunt of blood from the left to the right atrium. This will result in an equalization in the amount of blood entering both the left and right ventricles which effectively eliminates the normally wide splitting that inspiration typically causes in hearts without an atrial septal defect

Transient asymmetric ventricular septal hypertrophy in the

Aktuelle Kollektion zu Spitzenpreisen. Kostenlose Lieferung möglic The presence of asymmetric septal hypertrophy in an unoperated newborn patient with complete atrioventricular canal defect is most unusual and has not been reported, to the best of our knowledge. We describe a patient with Down syndrome in whom nonobstructive asymmetric septal hypertrophy was documented in the immediate newborn period and has. Conclusion: Asymmetrical septal hypertrophy was the commonest echocardiographic change found among IDMs, and HCM (Concentric Hypertrophy) was the commonest echocardiographic change found among INDMs. Both maternal diabetes (whatever its type) and macrosomia found to be risk factors for septal hypertrophy Asymmetrical septal hypertrophy is evident, with limited areas of LGE at the right ventricular junction (red arrows). E , CMR long-axis image from a severely symptomatic 27-year-old male HCM patient with the MYH7 mutation Arg694Cys (NM_000257.2 c.2080C>T), massive LV hypertrophy, small left ventricular (LV) cavity size, and dynamic outflow. Similarly the fetal heart, rich in insulin receptors, may undergo increased ventricular septal thickening in the presence of high insulin levels, culminating in asymmetric septal hypertrophy , , . Despite tight control of maternal blood glucose levels in gestational diabetics, asymmetric septal hypertrophic cardiomyopathy (HCM) is still.

Henry WL, Clark CE, Epstein SE. Asymmetric septal hypertrophy. Echocardiographic identification of the pathognomonic anatomic abnormality of IHSS. Circulation. 1973 Feb; 47 (2):225-233. [Google Scholar] Shah PM, Gramiak R, Adelman AG, Wigle ED. Role of echocardiography in diagnostic and hemodynamic assessment of hypertrophic subaortic stenosis These babies have asymmetric septal hypertrophy with secondary left ventricular outflow tract obstruction resembling idiopathic hypertrophic subaortic stenosis. It correlates with poor maternal control resulting in fetal hyperinsulinemia. Digoxin is contraindicated; the drug of choice for symptomatic babies in congestive failure is propranolol Septal thickness is often 4-6 mm more than normal. Asymmetric septal hypertrophy with absolute thickness more than 15 mm, septal / posterior wall (PW) ratio > 1.3 in normotensives and > 1.5 in hypertensives are some of the features. Rarely normal septal thickness can occur in genotype +ve cases, especially in cardiac troponin T mutations

Asymmetric Septal Hypertrophy in Appropriate for

  1. ant condition and involves gamma (γ)-subunit of AMP-activated protein kinase (AMPK) [4]. Left ventricular hypertrophy is due to glycogen deposition
  2. Medical Journal of Dr. D.Y. Patil University (2016-01-01) . Asymmetrical septal hypertrophy and hypertrophic cardiomyopathy in infant of diabetic mother: A reversible cardiomyopath
  3. We have not seen asym- metric septal hypertrophy or abnormal mitral or aortic valve motion on the echocardiograms of infants with ductus arteriosus alone, nor has this been mentioned in other studies of these infants.18-1'~ Allen and associates~' have reported asymmetric septal hypertrophy in neonates with a variety of congenital heart defects.
  4. ed heart muscle disease most often (60 to 70 percent) caused by mutations in one of several sarcomere genes which encode components of the contractile apparatus of the heart. (See Hypertrophic cardiomyopathy: Gene mutations and clinical genetic testing .
  5. This thickening can obstruct blood flow, cause a leaky heart valve, and result in irregular heart rhythms. Hypertrophic cardiomyopathy is also known as idiopathic hypertrophic subaortic stenosis (IHSS) or asymmetric septal hypertrophy (ASH). 3. Restrictive cardiomyopathy: This is the least common type of cardiomyopathy in children. It is.

Asymmetric septal hypertrophy in an infant exposed to

My 6 month old boy was just diagnosed with WPW syndrome and asymmetric septal hypertrophy. We discovered this after I noticed a prolonged period of lethargy and a fast heart rate. His EKG shows an abnormal P wave. After receiving an echo we were told there is no obstruction, but a definite thickening of the septum wall Decades ago, HCM was written about and known as idiopathic hypertrophic subaortic stenosis (IHSS) or asymmetrical septal hypertrophy (ASH). These terms were replaced by hypertrophic cardiomyopathy, because the segmental hypertrophy can occur in any segment of the ventricle, not just the septum Using a hospital-grade double electric breast pump will help you make more milk. Plan on pumping every 2-3 hours for the first two weeks. It is important to pump at least once in the middle of the night. At first, you will get very small amounts of colostrum but this milk is important for your baby's immune system, so save every drop Asymmetric septal hypertrophy (ASH) is seen in a number of cardiac malformations and myocardial diseases as well as in HCM, so it may be premature to settle on this diagnosis. Further review of the tape, however, notes the presence of systolic anterior motion (SAM) of the mitral valve, a finding that is highly specific (although not very.

Frequency of Asymmetrical Septal Hypertrophy in Large for

ASH - Asymmetric Septal Hypertrophy. Looking for abbreviations of ASH? It is Asymmetric Septal Hypertrophy. Asymmetric Septal Hypertrophy listed as ASH. Up to 40% of newborn infants of women with type 1 diabetes have echocardiographic signs of cardiomyopathy with cardiac enlargement and asymmetric septal hypertrophy. Most often ventricular hypertrophy, asymmetrical septal hypertrophy and outlow tract obstruction with cardiac dysfunction, the use of β-adrenergic blockers have been found to be useful. We report a case of a Nigerian neonate that was diagnosed with pathological left ventricular hypertrophy, asymmetrical septal hypertrophy and outlow tract obstruction an

Cardiac septal hypertrophy in hyperinsulinemic infants

Hypertrophic cardiomyopathy in infants: clinical features

I42.2 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2021 edition of ICD-10-CM I42.2 became effective on October 1, 2020. This is the American ICD-10-CM version of I42.2 - other international versions of ICD-10 I42.2 may differ. Applicable To Furthermore, newborns from pregnancies where glucose levels have been tightly controlled are still at risk of septal hypertrophy [11-13]. More surprisingly, newborns of tightly controlled diabetic mothers still have a high incidence of cardiac septal hypertrophy despite having abnormally low glycosylated fetal hemoglobin ( ) indicating lower.

Asymmetric septal hypertrophy, definition, causes

The hypertrophy is generally asymmetric and may be associated with obstruction of the ventricular outflow tract. A form of cardiac muscle disease, characterized by left and/or right ventricular hypertrophy (hypertrophy, left ventricular; hypertrophy, right ventricular), frequent asymmetrical involvement of the heart septum, and normal or. A: Parasternal long-axis view showing asymmetric septal hypertrophic cardiomyopathy in end diastole B: 4-chamber view showing a combination of basal and midventricular hypertrophic cardiomyopathy and an apical aneurysm (*) Ao: aorta. LA = left atrium. LV: left ventricle. RA = right atrium. RV = right ventricl Hypertrophic cardiomyopathy is also known as hypertrophic obstructive cardiomyopathy, HOCM; asymmetric septal hypertrophy, ASH; or idiopathic hypertrophic subaortic stenosis, IHSS. In this type of cardiomyopathy, the muscle mass of the left ventricle of the heart is larger than normal, or the wall between the two ventricles (septum) becomes. SUMMARY Twelve newborn infants of poorly-controlled diabetic mothers were transferred from IHSS:17 myocardial hypertrophy with asymmetrical septal hypertrophy, vigorous contraction, and increasedejectionfractionor %SID,reducedLVET, and mild systolic anterior movement of the mitra Asymmetric septal hypertrophy also called Familial hypertrophic cardiomyopathy is an inherited heart condition by thickening (hypertrophy) of the heart (cardiac) muscle. Thickening usually occurs in the interventricular septum, which is the muscular wall that separates the lower left chamber of the heart (the left ventricle) from the lower.

consistent with obstructive cardiomyopathy. A,Newborn echocandiognam. Abnormal thickening ofinterventnicular septum(5)andabnormal septal-to-freewallratio(7.5/3.1 withnormal lessthan 1.3/1) diagnostic ofasymmet-nicalseptal hypertrophy. B,Repeat echocandiogram at2#{189}months. Normal septal-to-postenion leftventricular wallratio = 4mm/4mm Hypertrophic cardiomyopathy is the most common cause of ventricular septal regional thickening, and it can mimic the symptoms of an intramural cardiac tumor through the cardiac MRI with T1 iso-intensity, T2 slightly hypo-intensity and no signal change after fat suppression imaging, which were the characteristics of fibroma due to its fibrous. The baby was given intravenous. hydrocortisone 5 2D echocardiogram on day 37 showing asymmetric septal hypertrophy and systolic anterior movement of the anterior mitral leaflet (arrow). Dexamethasone induced cardiac hypertrophy in newborn rats is accompanied by changes in myosin heavy chain phenotype and gene transcription

This effect is proportionate with the severity of septal hypertrophy. A disproportionally hypertrophic IVS in utero is an anabolic response due to fetal hyperinsulinemia caused by maternal hyperglycemia, which could be directly affected by the tightness of glycemic control. 14 , 15 Furthermore, Vural et al 16 stated that symptomatic HCM affects. Hypertrophic cardiomyopathy (HCM) is a heterogeneous disorder characterized by thickening of the heart and an increased incidence of sudden death. This study is aimed to determine the genetic cause of severe cardiac hypertrophy in an infant. An infant was assigned a diagnosis of ventricular preexcitation and severe biventricular HCM requiring septal myectomy

Hypertrophic cardiomyopathy (HCM) is a complex type of heart disease that affects the heart muscle. It causes thickening of the heart muscle (especially the ventricles, or lower heart chambers), left ventricular stiffness, mitral valve changes and cellular changes. Thickening of the heart muscle (myocardium) occurs most commonly at the septum Click for pdf: cyanotic congenital heart disease Introduction to Cyanosis Cyanosis is a bluish or purple discoloration of the skin and mucous membranes associated with poor oxygenation. It is noticeable when >5 g/dL of deoxygenated hemoglobin is present and usually assessed by pulse oximetry. It is very difficult to detect unless the arterial saturation is [

Hypertrophic cardiomyopathy (HCM) is a progressive myocardial disease where excessive ventricular wall thickening occurs. HCM is also one of the most common inherited cardiac disorders; a genetic cause can be identified in 35-45 % of HCM patients overall, and this increases to 60-65 %, when family history is positive for HCM [].This case series of a family with HCM points to the importance. Hypertrophic cardiomyopathy has been well documented in infants of diabetic mothers (IDMs). If this asymmetric septal enlargement is an anabolic result of fetal hyperinsulinemia triggered by maternal hyperglycemia during the third trimester, maternal glycosylated hemoglobin (HbA 1) levels, an indicator of glycemic control, should then correlate positively at delivery with newborn ventricular. Although HCM is typically characterized by asymmetric septal hypertrophy (ASH), almost any myocardial segment may be involved. The following two-dimensional (2D) echocardiographic criteria are used to aid diagnosis: However, genotype positive adults (including those who die suddenly) may have normal or near normal wall thickness Asymmetric septal hypertrophy andhypothyroidism in children ofthe hypothyroidism before cardiac function was studied.4 Oftheir four cases with congenital hypo-thyroidism, three were less than 6 months of age, while the fourth was 1 year old. Ourfirst case had clearly been hypothyroid from birth (11 months) and in the second case hypothyroidism.

The latter is also known as asymmetric septal hypertrophy or idiopathic hypertrophic subaortic stenosis. A transient form of dynamic obstruction of the left outflow tract is seen in infants of diabetic mothers, and is probably the consequence of fetal hyperglycemia and hyperinsulinemia Hypertrophy of the left ventricle may be concentric, asymmetric septal or apical. Hypertrophy pattern influences the ECG findings. ECG abnormalities that may be observed in hypertrophic cardiomyopathy The ECG above belongs to a 12 days-old newborn with hypertrophic cardiomyopathy (septal involvement) LV hypertrophy is a normal physiologic response to pressure and volume overload. Like any muscle, the heart grows bigger when it is forced to pump harder. When and How to Sleep Train Your Baby. It is likely that these abnormal Q waves are related to gross septal hypertrophy rather than myocardial infarction. Although the voltage criteria for left ventricular hypertrophy in the precordial leads were usually satisfied, there was no correlation between the height of RV 5 or the sum of RV 5 and SV 1 and the magnitude of the systolic. Asymmetric Septal Hypertrophy (ASH)- Interventricular septum thicker than LV free wall Cardiomyocyte hypertrophy and myofiber disarray * Dyspnea on exertion: arrhythmia, sudden death (most common cause of sudden death in young adolescents/athletes)

Asymmetrical Septal Hypertrophy can produce deep and narrow (dagger-like) Q waves in the lateral and inferior leads. LV Diastolic dysfunction (from a stiffened left ventricle) may cause Left Atrial hypertrophy, with signs of enlargement (P mitrale) on the ECG. There is some association between HCM and WPW syndrome, so a delta wave may be seen Within 48 h post delivery, echocardiograms were carried out on the newborn infants to identify those with hypertrophic cardiomyopathy, particularly asymmetrical septal hypertrophy. Results: The numbers of patients with abnormal UARI were similar in both the diabetic and control groups Define asymmetrical septal hypertrophy. asymmetrical septal hypertrophy synonyms, asymmetrical septal hypertrophy pronunciation, asymmetrical septal hypertrophy translation, English dictionary definition of asymmetrical septal hypertrophy. Olvera-Lopez, Asymmetrical septal hypertrophy in newborn infants of diabetic mothers, American. Pathological left ventricular hypertrophy, asymmetrical septal hypertrophy and outflow tract obstruction is a rare but known cardiac comorbidity in infants of diabetic mothers. The severity of this condition in IDMs can vary from an incidental finding on echocardiography to an infant with severe symptoms of congestive heart failure and specific.

TheEchoWeb - Patient Diagnosed With Asymmetric Septal

Infants of Diabetic Mothers Learn Pediatric

Hypertrophic Cardiomyopathy in Infants and Children

Hypertrophic cardiomyopathy - Symptoms and causes - Mayo

Hypertrophic Cardiomyopathy (HCM) • LITFL • ECG Library

Asymmetrical septal hypertrophy and hypertrophic

Asymmetric Septal Hypertrophy - DoveMe

  1. Sound with frequency of 20hz which is the upper limit of human hearing. Echocardiogram. Test that evaluates the internal structures and motions of the heart and great vessels with the use of ultrasound. Ultrasound transducer. Device that converts electrical energy to ultrasound energy
  2. Asymmetric Septal Hypertrophy. What is Coarctation of the aorta? A critical congenital heart defect. When does Coarctation of the aorta occur? When a baby's aorta does not form correctly as the baby grows and develops during pregnancy. YOU MIGHT ALSO LIKE... 84 terms. ARDMS Echo
  3. ant syndrome characterized by hypertelorism, downward slanting of the palpebral fissures, ptosis, low‐set posteriorly angulated ears, short stature, and congenital heart disease, most commonly pulmonary valve stenosis, hypertrophic cardiomyopathy (HCM), and atrial septal defects (ASDs). We report a 6‐day‐old girl who had an.

Ventricular septal defect. Q21.0 is a billable/specific ICD-10-CM code that can be used to indicate a diagnosis for reimbursement purposes. The 2021 edition of ICD-10-CM Q21.0 became effective on October 1, 2020. This is the American ICD-10-CM version of Q21.0 - other international versions of ICD-10 Q21.0 may differ Edward Hurley, MD, specializes in general cardiology and interventional cardiology. He performs cardiac catheterization and coronary interventions at Chesterfield Cardiology Care. Dr. Hurley earned his medical degree at the University of Missouri. He completed his residency at the Mayo Clinic, his fellowship in cardiovascular medicine at the University of Louisville and his fellowship in. The newborns of the diabetic mothers were classified according to septal thickness as group 1 (16 newborns with septal hypertrophy) or group 2 (31 newborns without septal hypertrophy). The study group consisted of three cohorts: groups 1, 2, and 3 (control group)

Cardiovascular - Cardiac Pathology - Pathology with PathHypertrophic cardiomyopathy

  1. Asymmetric Septal Hypertrophy (ASH) in Infancy Circulatio
  2. Asymmetric Septal Hypertrophy in Infants of Diabetic
  3. Familial hypertrophic cardiomyopathy Genetic and Rare

Hypertrophic cardiomyopathy - neonatal Radiology Case

  1. Pathological left ventricular hypertrophy and outflow
  2. Assessment of cardiomyopathy in fetuses of women with
  3. Prevalence of Hypertrophic Cardiomyopathy in Fetuses of
  4. Hypertrophic cardiomyopathy - Wikipedi

Neonatal Cardiomyopathies Thoracic Ke

  1. Hypertrophic Cardiomyopathy (HCM) • LITFL • ECG Library
  2. pedspackratscombo:) Flashcards Quizle
  3. Asymmetric bei Amazon
  4. Unusual association of hypertrophic cardiomyopathy with
  5. Study of Echocardiographic Changes in Macrosomic Neonate
Distinguishing ventricular septal bulge versusLipomatous hypertrophy of the interatrial septum: aAssociated professor Masyuta DDr